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database / hormone

Cetrorelix

approved hormone reproductiveendocrine sequence not characterised
ClOOOOOOOOOOOOOONNNNNNNNNNNNNNNNNHHHHHHHHHHHHHHHHHHHH
102 heavy atoms · 199 bonds · drag to pan, scroll to zoom C70N17O14Cl1

Skeletal structure drawn from the computed atomic coordinates in PubChem CID 25074887. Carbons are implicit vertices; hydrogens on carbon are suppressed, as in any structural formula. Nothing here is estimated — every atom sits where PubChem placed it.

Research reference only. The observations below are recorded in the cited literature. Nothing here is advice or a recommendation, and no dosing information is published on this site.

Sequence

Primary structure not characterised in the public records this index draws on. Nothing is shown rather than something invented.

GnRH antagonist built from multiple non-proteinogenic and D-amino acids.

Molecular data

Chemical fields come from the PubChem compound record; computed values are derived from the sequence shown above.
molecular formulaC70H92ClN17O14
molecular weight1431 Da (PubChem)
computed backbone massnot characterised
lengthnot characterised
net charge (pH 7.4)not characterised
mean hydropathynot characterised
half-lifenot characterised
delivery routesubcutaneous
PubChem CID25074887
PDBnot characterised
UniProt parentnot characterised

Mechanism

Competitive GnRH receptor antagonist; suppresses LH without an initial flare.

Reported targets: GnRHR

Experimental structure

No experimental structure of this peptide is deposited in the PDB. Nothing is rendered here — a predicted fold would not be a structure, and this site does not draw one.

Reported effects

Each row is an outcome described in the literature indexed for this peptide. Reported in the cited work — not a claim, not a recommendation.

Reported observations and the corpus they are drawn from.
reported outcomewhere it appears
LH suppressionA new cetrorelix-based estrogen-free ovarian synchronization protocol for fi… Theriogenology 2026
assisted-reproduction outcomes in trialsCetrorelix suppresses the dominant follicle and synchronizes follicular wave… Biology of reproduction 2026

Registered trials

Records from ClinicalTrials.gov. Listed for reference — this project sponsors no trial and is not involved in any of them.
NCTtitlestatusphase
NCT04157725Mild Stimulation Protocol Using Clomiphene Citrate for Women With PCOS Undergoing in Vitro FUNKNOWNPHASE4
NCT02496754Follicular Long GnRH Agonist Versus Antagonist Protocol in PCOS Women Undergoing in Vitro FeCOMPLETEDNA
NCT07736261Drospirenone-Primed Ovarian Stimulation Versus GnRH Antagonist Protocol in Poor Ovarian RespNOT_YET_RECRUITINGPHASE1, PHASE2
NCT04854707An Observational Study of Follitropin Alpha Biosimilar: the Real-world DataCOMPLETED
NCT02821819Random-start Ovarian Stimulation in Egg-donors (ROSE)TERMINATEDPHASE4

Literature (8)

A new cetrorelix-based estrogen-free ovarian synchronization protocol for fixed-time artificial insemination in beef cattle — Theriogenology 2026

abstract not reproduced — this record is not open-access, so it is linked rather than copied.

publisher record ↗ · PMID 41864162 · DOI 10.1016/j.theriogenology.2026.117904

Cetrorelix suppresses the dominant follicle and synchronizes follicular waves and ovulation in cattle† — Biology of reproduction 2026

To determine the effects of a GnRH antagonist on ovarian function in cattle, we tested the hypotheses that cetrorelix will (1) cause regression of the extant dominant follicle by altering gonadotrophin secretion and (2) induce a new follicular wave emergence at a consistent time after treatment. In Experiment 1, heifers were given 1.5 mg cetrorelix im on Days 1-2 (Day 0 = wave emergence), Days 3-4, and Days 6-7, or normal saline (Control, n = 8 per group). The dominant follicle was smaller and regressed earlier in groups treated with cetrorelix on Days 1-2 or Days 3-4 than in Controls (P = 0.01). Plasma LH concentrations were lower (P = 0.04) for 3 days after treatment, and the pre-wave surge in FSH occurred earlier in groups treated with cetrorelix on Days 1-2 or Days 3-4 than Controls (P = 0.01). Corpus luteum diameter was smaller (P = 0.01) in the cetrorelix groups, but luteal vascula…

open access ↗ · PMID 41395811 · DOI 10.1093/biolre/ioaf276

GnRH Agonists and Antagonists in IVF/ICSI Cycles of PCOS Women: A Network Meta-Analysis — Journal of the College of Physicians and Surgeons--Pakistan : JCPSP 2026

abstract not reproduced — this record is not open-access, so it is linked rather than copied.

publisher record ↗ · PMID 42152207 · DOI 10.29271/jcpsp.2026.05.654

Efficacy and safety of different cetrorelix doses in the luteal phase for preventing ovarian hyperstimulation syndrome: A cross-sectional study — International journal of reproductive biomedicine 2026

BackgroundOvarian hyperstimulation syndrome (OHSS) is a severe iatrogenic disorder resulting from controlled ovarian stimulation. The administration of a gonadotropin-releasing hormone antagonist (cetrorelix) during the luteal phase is a practical approach for preventing OHSS.ObjectiveThis study aimed to evaluate the efficacy and safety of different cetrorelix regimens for preventing OHSS in high-risk groups.Materials and methodsIn this cross-sectional study, data of 271 women with a high-risk profile for OHSS presented to the Infertility Center of Kosar hospital, Urmia, Iran from March 2023 and March 2025 and those who underwent intracytoplasmic sperm injection were extracted from their medical records. Participants were given prophylactic luteal-phase cetrorelix regimen of [dose redacted] twice daily (BID group, n = 101), 0.5 mg 3 times daily (TDS group, n = 38), or 0.75 mg TDS group (n = 102).…

open access ↗ · PMID 42312112 · DOI 10.18502/ijrm.v24i4.21173

Cetrorelix promotes cell apoptosis via the PI3K-AKT-FOXO1 pathway in epithelial ovarian cancer — Frontiers in oncology 2025

IntroductionEpithelial ovarian cancer (EOC) has a dismal prognosis, and recent therapeutic advancements have been limited. The aim of our study was to clarify the role and mechanism of cetrorelix in EOC apoptosis and to evaluate the clinical relevance of GnRHR, AKT, and FOXO1 in EOC patients.MethodsApoptosis was assessed using flow cytometry, Hoechst staining, and Western blotting. FOXO1, p-AKT and GnRHR knockdown via siRNA was performed to reverse cetrorelix-induced apoptosis. Mechanistic insights were explored using apoptosis gene PCR arrays, qRT-PCR, and Western blotting. In vivo efficacy was tested in a xenograft mouse model. Immunohistochemistry (IHC) was used to assess GnRHR, AKT,p-AKT and FOXO1 expression in EOC tissues, and survival analysis was performed using Kaplan-Meier and Cox regression analyses.ResultsCetrorelix facilitated EOC apoptosis both in vitro and in a xenograft mo…

open access ↗ · PMID 41458598 · DOI 10.3389/fonc.2025.1631576

Comparison of Chitosan-Poloxamer Nanoparticles and Poloxamer-based In-Situ Forming Gel for Nose-to-Brain Delivery of Cetrorelix: In Vitro and Pharmacokinetic Studies in Rats — AAPS PharmSciTech 2026

abstract not reproduced — this record is not open-access, so it is linked rather than copied.

publisher record ↗ · PMID 42243608 · DOI 10.1208/s12249-026-03430-6

Effect of a GnRH antagonist on the fate of the dominant ovarian follicle and the emergence of the next follicular wave in alpacas — Theriogenology 2026

abstract not reproduced — this record is not open-access, so it is linked rather than copied.

publisher record ↗ · PMID 41248592 · DOI 10.1016/j.theriogenology.2025.117733

Synthesis of Diastereomerically Pure Cetrorelix Acetate by Using Fmoc Solid-Phase Peptide Synthesis (SPPS) Strategy: A Commercially Viable Approach — Journal of peptide science : an official publication of the European Peptide Society 2025

abstract not reproduced — this record is not open-access, so it is linked rather than copied.

publisher record ↗ · PMID 40386934 · DOI 10.1002/psc.70030

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